HIV -- to Oufwit

In one recent book by Leanne K. Currie-McGhee (2008) that “HIV replicates itself by inserting its RNA into a cell’s DNA. When the cells divide, a new copy of the Virus is produced. This increases the amount of HIV in a person. HIV then attacks CD4 cells, also known as T Cells. T Cells are immune cells that fight off illnesses. When HIV hijacks these cells, the body loses its ability to fight off illnesses. AZT blocks the replication of HIV. As a result, AZT decreases the amount of HIV in the body and increases a person’s healthy CD4 cell count.” While this is a good summary of the operational dynamics of HIV, it hardly tells anything about it, and does not much good to any scientist interest in exploring new ideas. From her book we learn the current classes of HIV medications; Non-nucleoside reverse transcriptase (NNRTIs), (2) Non-nucleoside reverse transcriptase inhibitors (NRTIs), (3) Protease Inhibitors and (4) Fusion Inhibitors, all of which work to perform one function stop HIV multiplication. She then adds the struggling function of the various groups of medication that “NNRTIs and NRTIs are both reverse transcriptase inhibitors that stop viral RNA from being turned into DNA. The nucleoside and non-nucleoside reverse transcriptase inhibitors target slightly different parts of the enzyme. Protease inhibitors prevent the HIV Virus from forming mature Virions. Fusion inhibitors stop HIV from actually entering CD4 cells.” Such summary of the whole process is acceptable in understanding the current medication with Virus, but does not nearly open the main issue associated with the whole Hot Topic. In reality, there is more to be desired about the patient to patient experience which HIV communities can offer, for instance the impact of new but also old drugs, and why these drugs offer a future that encourages replications if not control at Virions level. Put it bluntly, the emphasis on cocktails and combination are attempt at final case of the HIV patient, whereas the main attention is not only the mode of transmission or pathology, rather, why HIV virus only affect human beings.